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Ginkgo B

Ginkgo B

By: MRM

Increases Blood Flow To The Brain & Surrounding Tissues!

Ginkgo Biloba has been used medicinally for thousands fo years. Today, clinical research has substantiated the amazing biological effects of Ginkgo by establishing it's ability to increase blood flow to the brain and peripheral tissues. This means greater oxygen and vital nutrients to these areas.

Availability: In stock

Size/Flavor Retail Savings M&S Price Qty
120 Vegetarian Caps $19.99 51%
$9.99

Ginkgo B Product Details

Ginkgo Biloba has been used medicinally for thousands fo years. Today, clinical research has substantiated the amazing biological effects of Ginkgo by establishing it's ability to increase blood flow to the brain and peripheral tissues. * This means greater oxygen and vital nutrients to these areas. * Of the active compounds, ginkgolide B has been shown to be the most potent and effective. MRM offers you the highest percentage of ginkgolide B with an outstanding minimum of 3.0% for maximum benefits. *

More About Ginkgo Biloba:

Ginkgo Biloba is an extract from cultivated leaves of the Ginkgo Tree. As one of the oldest living trees on the planet, Ginkgo's medicinal use can be traced back through Chinese medical literature to 2800 BC. Ginkgo leaves have been used in traditional Chinese medicine for "their ability to benefit the brain and more". Ginkgo leaf extracts are now one of the leading prescription medicines in both France and Germany. Research has shown the active ingredients in ginkgo biloba to be a class of compounds known as ginkgolides. Of this class, ginkgolide B, referred to in many investigations as BN52021, is the most researched ginkolide and has shown to be the most therapeutic (1).

How Ginkgo Works: Platelets, irregular shaped discs in the blood, are responsible for the clotting of wounds. Platelet-activating factor (PAF) is released by the platelets causing them to stick together. Studies have indicated that a component of ginkgo biloba, ginkgolide B, is a potent platelet-activating factor (PAF) antagonist (2,3).

References:

  1. Lancet. 1:248, 1987.
  2. J Ethnopharmacol. 50:131, 1996.
  3. Drugs of the Future, 12; No 7, 1987.